Displaying items by tag: Division of Cardiovascular Disease
The 11 postdocs selected to receive Outstanding Postdoctoral Awards represent nine departments, divisions and schools across UAB.
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UAB Medicine has a long history of caring for patients with adult congenital heart disease, and the Alabama Adult Congenital Heart Program is the only one in Alabama and one of a handful in the country that provide continuing care for patients as they move into adulthood.
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A program that supports historically disadvantaged health professionals chooses two researchers in the UAB Department of Medicine as finalists.
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A fatty acid-derived bioactive molecule called lipoxin improved heart function after a heart attack, as the lipoxin prompted early activation of the resolving phase of the immune response.
Knowledge from this study and others may help physicians boost healing and prevent heart failure in patients after a heart attack.
These leaders represent the next generation of cardiologists who are working to educate their colleagues, conduct research on new and novel strategies to advance cardiovascular care, and innovate the unique aspects of the cardiology specialty.
Twelve School of Medicine faculty were recently honored as winners of the UAB Graduate Dean's Excellence in Mentorship Award.
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- school of medicine
- department of pharmacology and toxicology
- department of medicine
- division of infectious diseases
- division of gerontology, geriatrics, and palliative care
- division of pulmonary, allergy and critical care medicine
- division of nephrology
- division of cardiovascular disease
- department of pediatrics
- division of pediatric infectious diseases
- department of surgery
- division of cardiothoracic surgery
- department of pathology
- department of neurobiology
- department of biomedical engineering
The grant will initially fund two fellowship positions for postdoctoral fellows (basic or clinical) wishing to train in the area of heart failure research.
A heart attack can happen even if to those who are relatively young and fit, and genetics can play a role.
The findings suggest targeting specific T-cell subsets may be a therapeutic approach to prevent heart failure after a heart attack.