Assistant Professor of MedicineXiaoli-Li

Address:
 

Shelby Bldg, Room 205
1825 University Blvd
35294-2182

Telephone: (205) 996-4477
Fax: (205) 975-4404
Email: xili@uabmc.edu
 

Education


Diploma, Wuhan University, China, 1979
MS, Wuhan University, China, 1982
PhD, University of Alabama at Birmingham, 1992
Post-Doctoral Training (Immunology): Cleveland Clinic Foundation, 1992-1994

Post-Doctoral Training: University of Alabama at Birmingham, 1995-1997

Research Description

 
Dr. Li's major research interest is investigation of biological effects of Fcγ Receptor polymorphisms. Elucidation of the biological functions mediated by Fcγ Receptors.
 

Publications

Click here for a more complete list of publications. Below are a few selected papers.


Li X, Baskin JG, Mangan EK, Su K, Gibson AW, Ji C, Edberg JC, Kimberly RP. 2012. The unique cytoplasmic domain of human FcγRIIIA regulates receptor-mediated function. J Immunol. 189(9):4284-94.  PMC3478424 PMID:23024279 


Su K, Yang H, Li X, Li X, Gibson AW, Cafardi JM, Zhou T, Edberg JC, Kimberly RP. 2007. Expression profile of FcgammaRIIb on leukocytes and its dysregulation in systemic lupus erythematosus. J Immunol. 178(5):3272-80. PMID: 17312177

 
Su K, Li X, Edberg JC, Wu J, Ferguson P, Kimberly RP. 2004. A promoter haplotype of the immunoreceptor tyrosine-based inhibitory motif-bearing FcgammaRIIb alters receptor expression and associates with autoimmunity. II. Differential binding of GATA4 and Yin-Yang1 transcription factors and correlated receptor expression and function. J Immunol. 172(11):7192-9. PMID: 15153544

 
Su K, Wu J, Edberg JC, Li X, Ferguson P, Cooper GS, Langefeld CD, Kimberly RP. 2004. A promoter haplotype of the immunoreceptor tyrosine-based inhibitory motif-bearing FcgammaRIIb alters receptor expression and associates with autoimmunity. I. Regulatory FCGR2B polymorphisms and their association with systemic lupus erythematosus. J Immunol. 172(11):7186-91.

 

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