Greg Williams
| This email address is being protected from spambots. You need JavaScript enabled to view it.Research Editor, UAB News
(205) 975-3914
gdw@uab.edu
Williams is responsible for helping to further burnish the national reputation of the UAB basic science enterprise, as well as for promoting novel research efforts across all university disciplines. He develops news and feature content for delivery through print and online media, and engages with social media communities formed around common interests relevant to the UAB AMC 21 Research Strategic Plan. Williams is a graduate of the Medill School of Journalism at Northwestern University, and previously worked as senior science editor at the University of Rochester Medical Center in New York State.
Beats include: Biochemistry and Molecular Genetics, Biomedical Engineering, Cell Biology, Microbiology, Physiology and Biophysics, UAB Research Foundation
(205) 975-3914
gdw@uab.edu
Williams is responsible for helping to further burnish the national reputation of the UAB basic science enterprise, as well as for promoting novel research efforts across all university disciplines. He develops news and feature content for delivery through print and online media, and engages with social media communities formed around common interests relevant to the UAB AMC 21 Research Strategic Plan. Williams is a graduate of the Medill School of Journalism at Northwestern University, and previously worked as senior science editor at the University of Rochester Medical Center in New York State.
Beats include: Biochemistry and Molecular Genetics, Biomedical Engineering, Cell Biology, Microbiology, Physiology and Biophysics, UAB Research Foundation
Newfound details of rare disease may advance understanding of brain tumors.
A genetic variant that shapes each person’s immune response contributes to the chances of developing autoimmune disease.
Changes to a key protein amplified its natural ability to counter kidney disease, according to a study published today by School of Medicine researchers in the journal Nature Medicine.
Damage to heart muscle caused by a heart attack turns on immune cells in the spleen that accelerate heart failure.
Fast, inexpensive gene sequencing needed to make possible personalized medicine.
Mitochondrial DNA determines oxidant levels and may further explain why some people get sick and others don’t.
From The Mix: A recent study found that, along with triggering inflammation, TNF-alpha also blocks the engulfment and removal of dying cells.
A new study found chronic alcohol use may interfere with the genetic clocks in liver cells to accelerate liver damage.
Study is first to reveal the role of the kidney in the regulation of iron metabolism following injury.
As part of the award, Peter Anderson, D.V.M., Ph.D., director of Pathology Undergraduate Education, will lead faculty development programs and hands-on workshops at the Tzu-Chi University College of Medicine in Taiwan for two weeks.