Displaying items by tag: heart disease

Overexpression of CCND2 increased growth and division of grafted heart muscle cells, resulting in better heart function and decreased size of dead tissue.
Knowledge from this study and others may help physicians boost healing and prevent heart failure in patients after a heart attack.
The findings suggest targeting specific T-cell subsets may be a therapeutic approach to prevent heart failure after a heart attack.
Cardiac muscle patches in this proof-of-concept research may represent an important step toward the clinical use of 3-D-printing technology, as researchers have grown heart tissue by seeding a mix of human cells onto a 1-micron-resolution scaffold made with a 3-D printer.
A Western-style diet, with more omega-6 fatty acids than the Mediterranean, dysregulates lipid signaling in aged mice and promotes inflammation.
A UAB researcher will examine the underlying environmental impact on genes of Mexican Americans in relation to cardiometabolic syndrome, a disorder caused by several interrelated risk factors that can lead to heart disease or Type 2 diabetes.
The collaboration among UAB, University of Wisconsin and Duke University will use bioengineered stem cells and bioengineered tissue to treat heart failure after heart attacks.
The zebrafish is making its mark in the field of biomedical research, becoming one of the fastest-growing models of human disease.
Springing forward with daylight saving time may increase your risk of having a heart attack if you have a history of heart disease.
UAB’s Virginia Howard has been honored for her work to find novel and powerful approaches to reduce the burden of stroke and heart disease.
Increased risk of major adverse cardiac events after the later surgery persists for one year.
The protective effect is achieved by reducing acute inflammation in the spleen and the left ventricle of the heart.
Research revealing new evidence about the role of the spleen following heart attack will be honored during the AHA scientific meeting Nov. 15-19.