Displaying items by tag: UAB Comprehensive Diabetes Center
Department of Psychology Associate Professor Michael Sloane, Ph.D., grew up in the rolling countryside of Ireland, where processed foods were far from a dietary staple– everything was farm to table. His family grew all their own vegetables and fruits, raised chickens for eggs, and bought meat from a neighboring farmer or fresh cuts from the town butcher. So, when he came over to the U.S. to complete his doctoral work at Northwestern University in 1979, Sloane was plunged into a consumer market that was riddled with highly processed and sugary food choices.
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The year was 1997. Department of Medicine Communications Director Carolyn Walsh was traveling on i459 when things did not feel quite right. She was fighting to stay awake at the wheel, noticing bouts of drowsiness on and off. Before she could pull over, Walsh was in a car accident.
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UAB Heersink School of Medicine staff council representatives have officially been named for the 2022-2024 term. See who has been elected to represent you!
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Three School of Medicine faculty projects were recently funded as AMC21 2019 Multi-PI awardees.
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Inhibition of this microRNA might improve response to newer diabetes drugs, such as Byetta, Victoza, Trulicity, Januvia, Onglyza and Tradjenta.
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A multidisciplinary research team at UAB looks to address obesity as it is genetically passed from mother to child.
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Recruitment for the trial continues as Myers discusses the benefits she received from participating and the knowledge she has gained to take control of her health care.
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Lead author of paper published in Diabetes Research and Clinical Practice journal says, while causal relationship cannot be inferred, findings are “absolutely encouraging.”
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Many patients diagnosed with type 1 diabetes as adults do not fit the standard profile for a diabetes diagnosis; they are younger, are relatively fit and have little if any belly fat.
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Three-year grant establishes consortium to better organize and support collaborative research related to the loss of functional beta cell mass in type 1 diabetes.
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