Publications Citing Core B

1.              Saigusa, T., R. Reichert, J. Guare, B.J. Siroky, M. Gooz, S. Steele, R.A. Fenton, P.D. Bell, and R.J. Kolb, Collecting duct cells that lack normal cilia have mislocalized vasopressin-2 receptors. Am J Physiol Renal Physiol, 2012. 302(7): p. F801-8.http://www.ncbi.nlm.nih.gov/pubmed/22205228

2.              Williams, C.L., C. Li, K. Kida, P.N. Inglis, S. Mohan, L. Semenec, N.J. Bialas, R.M. Stupay, N. Chen, O.E. Blacque, B.K. Yoder, and M.R. Leroux, MKS and NPHP modules cooperate to establish basal body/transition zone membrane associations and ciliary gate function during ciliogenesis. J Cell Biol, 2011. 192(6): p. 1023-41.http://www.ncbi.nlm.nih.gov/pubmed/21422230

3.              Tower-Gilchrist, C., E. Lee, and E. Sztul, Endosomal trafficking of the G protein-coupled receptor somatostatin receptor 3. Biochem Biophys Res Commun, 2011. 413(4): p. 555-60.http://www.ncbi.nlm.nih.gov/pubmed/21925142

4.              Sharma, N., Z.A. Kosan, J.E. Stallworth, N.F. Berbari, and B.K. Yoder, Soluble levels of cytosolic tubulin regulate ciliary length control. Mol Biol Cell, 2011. 22(6): p. 806-16.http://www.ncbi.nlm.nih.gov/pubmed/21270438

5.              Masyukova, S.V., M.E. Winkelbauer, C.L. Williams, J.N. Pieczynski, and B.K. Yoder, Assessing the pathogenic potential of human Nephronophthisis disease-associated NPHP-4 missense mutations in C. elegans. Hum Mol Genet, 2011. 20(15): p. 2942-54.http://www.ncbi.nlm.nih.gov/pubmed/21546380

6.              Fogelgren, B., S.Y. Lin, X. Zuo, K.M. Jaffe, K.M. Park, R.J. Reichert, P.D. Bell, R.D. Burdine, and J.H. Lipschutz, The exocyst protein Sec10 interacts with Polycystin-2 and knockdown causes PKD-phenotypes. PLoS Genet, 2011. 7(4): p. e1001361.http://www.ncbi.nlm.nih.gov/pubmed/21490950

7.              Egorova, A.D., P.P. Khedoe, M.J. Goumans, B.K. Yoder, S.M. Nauli, P. ten Dijke, R.E. Poelmann, and B.P. Hierck, Lack of primary cilia primes shear-induced endothelial-to-mesenchymal transition. Circ Res, 2011. 108(9): p. 1093-101.http://www.ncbi.nlm.nih.gov/pubmed/21393577

8.              Berbari, N.F., N.W. Kin, N. Sharma, E.J. Michaud, R.A. Kesterson, and B.K. Yoder, Mutations in Traf3ip1 reveal defects in ciliogenesis, embryonic development, and altered cell size regulation. Dev Biol, 2011. 360(1): p. 66-76.http://www.ncbi.nlm.nih.gov/pubmed/21945076

9.              Bell, P.D., W. Fitzgibbon, K. Sas, A.E. Stenbit, M. Amria, A. Houston, R. Reichert, S. Gilley, G.P. Siegal, J. Bissler, M. Bilgen, P.C. Chou, L. Guay-Woodford, B. Yoder, C.J. Haycraft, and B. Siroky, Loss of primary cilia upregulates renal hypertrophic signaling and promotes cystogenesis. J Am Soc Nephrol, 2011. 22(5): p. 839-48.http://www.ncbi.nlm.nih.gov/pubmed/21493775

10.           Zhou, J., X. Ouyang, X. Cui, T.R. Schoeb, L.E. Smythies, M.R. Johnson, L.M. Guay-Woodford, A.B. Chapman, and M. Mrug, Renal CD14 expression correlates with the progression of cystic kidney disease. Kidney Int, 2010. 78(6): p. 550-60.http://www.ncbi.nlm.nih.gov/pubmed/20555320

11.           Hellman, N.E., Y. Liu, E. Merkel, C. Austin, S. Le Corre, D.R. Beier, Z. Sun, N. Sharma, B.K. Yoder, and I.A. Drummond, The zebrafish foxj1a transcription factor regulates cilia function in response to injury and epithelial stretch. Proc Natl Acad Sci U S A, 2010. 107(43): p. 18499-504.http://www.ncbi.nlm.nih.gov/pubmed/20937855

12.           O'Connor, A.K., R.A. Kesterson, and B.K. Yoder, Generating conditional mutants to analyze ciliary functions: the use of Cre-lox technology to disrupt cilia in specific organs. Methods Cell Biol, 2009. 93: p. 305-30.http://www.ncbi.nlm.nih.gov/pubmed/20409823

13.           Kesterson, R.A., N.F. Berbari, R.C. Pasek, and B.K. Yoder, Utilization of conditional alleles to study the role of the primary cilium in obesity. Methods Cell Biol, 2009. 94: p. 163-79.http://www.ncbi.nlm.nih.gov/pubmed/20362090

14.           Williams, C.L., M.E. Winkelbauer, J.C. Schafer, E.J. Michaud, and B.K. Yoder, Functional redundancy of the B9 proteins and nephrocystins in Caenorhabditis elegans ciliogenesis. Mol Biol Cell, 2008. 19(5): p. 2154-68.http://www.ncbi.nlm.nih.gov/pubmed/18337471

15.           Willaredt, M.A., K. Hasenpusch-Theil, H.A. Gardner, I. Kitanovic, V.C. Hirschfeld-Warneken, C.P. Gojak, K. Gorgas, C.L. Bradford, J. Spatz, S. Wolfl, T. Theil, and K.L. Tucker, A crucial role for primary cilia in cortical morphogenesis. J Neurosci, 2008. 28(48): p. 12887-900.http://www.ncbi.nlm.nih.gov/pubmed/19036983

16.           Mrug, M., J. Zhou, Y. Woo, X. Cui, A.J. Szalai, J. Novak, G.A. Churchill, and L.M. Guay-Woodford, Overexpression of innate immune response genes in a model of recessive polycystic kidney disease. Kidney Int, 2008. 73(1): p. 63-76.http://www.ncbi.nlm.nih.gov/pubmed/17960140

17.           Olteanu, D., M.B. Hovater, and E.M. Schwiebert, Intraluminal autocrine purinergic signaling within cysts: implications for the progression of diseases that involve encapsulated cyst formation. Am J Physiol Renal Physiol, 2007. 292(1): p. F11-4.http://www.ncbi.nlm.nih.gov/pubmed/16940560

18.           Banizs, B., P. Komlosi, M.O. Bevensee, E.M. Schwiebert, P.D. Bell, and B.K. Yoder, Altered pH(i) regulation and Na(+)/HCO3(-) transporter activity in choroid plexus of cilia-defective Tg737(orpk) mutant mouse. Am J Physiol Cell Physiol, 2007. 292(4): p. C1409-16.http://www.ncbi.nlm.nih.gov/pubmed/17182727

19.           Siroky, B.J., W.B. Ferguson, A.L. Fuson, Y. Xie, A. Fintha, P. Komlosi, B.K. Yoder, E.M. Schwiebert, L.M. Guay-Woodford, and P.D. Bell, Loss of primary cilia results in deregulated and unabated apical calcium entry in ARPKD collecting duct cells. Am J Physiol Renal Physiol, 2006. 290(6): p. F1320-8.http://www.ncbi.nlm.nih.gov/pubmed/16396941