Jeff Hansen

Jeff Hansen

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Research Editor

jeffhans@uab.edu • (205) 209-2355

Communicates UAB research discoveries and initiatives from across the university for a variety of audiences.

Specific beats include: biochemistry; cell, developmental and integrated biology; microbiology; molecular genetics; neurobiology; pathology; pharmacology and tocixology; Alabama Drug Discovery Alliance; Bill L. Harbert Institute for Innovation and Entrepreneurship.

This NIH-funded conference is part of UAB’s effort to engage and retain neuroscience graduate students from underrepresented ethnic and racial groups across the United States.

This is the second potential diagnostic application for an investigational biomarker, and discussions are underway with industry partners to develop an assay from this UAB technology.

For a second summer, Maria Onatunde traveled to UAB from Florida to participate in UAB's PARAdiGM program, which offers in-depth research experience for undergraduates from disadvantaged and minority backgrounds.
An Iowa college student with dreams of being a physician gleaned invaluable experience and inspiration out of a short summer the intensive research and clinical environment offered through UAB’s Biomedical Sciences Undergraduate Research Program.
Altered excitability is seen in brain neurons in epilepsy, depression, drug addiction and other disorders, and this discovery may offer a potential therapeutic target.

Kelly Walters relishes her access to scientists, clinicians and research opportunities unavailable to undergraduates at many other universities as she builds toward a career in medicine.  

UAB researchers have discovered that an infant’s airway — once thought to be sterile until after birth — is colonized by bacteria or bacterial DNA, which could be protective for or predict development of severe lung disease, knowledge that may offer a therapeutic target.
The mechanism of widespread reorganization of DNA methylation may be a therapeutic target to prevent or reverse dyskinesia.
Chang, who was named a 2015 Fulbright Scholar in 2015, is this year one of 57 Phi Kappa Phi fellows selected nationwide.
Researchers have found that an interaction between a mutant gene and alpha synuclein in neurons leads to hallmark pathologies seen in Parkinson’s disease, findings that may lead to new mechanisms and targets for neuroprotection.
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