Displaying items by tag: department of microbiology
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The biomarker can identify beta cell death in children and adults with recent-onset Type 1 diabetes and in antibody-positive at-risk subjects before disease onset.
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- release
- school of medicine
- school of public health
- department of medicine
- department of microbiology
- department of surgery
- department of pathology
- department of biostatistics
- division of endocrinology diabetes and metabolism
- division of clinical immunology and rheumatology
- comprehensive diabetes center
Beta cells in the pancreas produce the body’s insulin, and their loss or dysfunction causes diabetes.
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Jessica Scoffield, Ph.D., is the first recipient of an underrepresented faculty research fellowship.
BK polyomavirus reactivation is a major source of kidney damage in transplant recipients leading to rejection, so reducing viral levels may save more kidneys.
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No current therapy for multiple sclerosis exists to halt the progression of this autoimmune disease.
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Tissue-resident B cells likely have important differences that affect autoimmunity and transplant rejection.
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- release
- school of medicine
- department of microbiology
- department of medicine
- department of surgery
- division of clinical immunology and rheumatology
- comprehensive transplant institute
- division of infectious diseases
- division of transplantation
- comprehensive arthritis musculoskeletal and autoimmunity center
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The pilots link two powerful research entities, to focus on asthma, vaccine development and improving safety of therapeutic drugs.
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The hygiene hypothesis says increased sanitation in industrialized countries has led to increases in asthma.
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A novel informatics technique allows researchers to identify different strains of the same bacterial species.
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Understanding the molecular basis of binding informs ways to prevent retroviral replication, including the HIV retrovirus.
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Up to now, it has not been clear if these cells might be useful to combat influenza infections or even if they exist at all.
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This finding may point to a therapeutic target to treat tuberculosis, which kills 1.3 million people each year.
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The mechanism the tuberculosis pathogen uses in its escape suggests possible therapies to improve patient outcomes.
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